Следене
Michael Pusch
Michael Pusch
Istituto di Biofisica, Genova, Italia
Потвърден имейл адрес: ge.ibf.cnr.it
Заглавие
Позовавания
Позовавания
Година
Mutation in the neuronal voltage-gated sodium channel SCN1A in familial hemiplegic migraine
M Dichgans, T Freilinger, G Eckstein, E Babini, B Lorenz-Depiereux, ...
The Lancet 366 (9483), 371-377, 2005
10442005
Rates of diffusional exchange between small cells and a measuring patch pipette
M Pusch, E Neher
Pflügers Archiv 411, 204-211, 1988
8061988
A chloride channel widely expressed in epithelial and non-epithelial cells
A Thiemann, S Gründer, M Pusch, TJ Jentsch
Nature 356 (6364), 57-60, 1992
6691992
Mapping the site of block by tetrodotoxin and saxitoxin of sodium channel II
H Terlau, SH Heinemann, W Stühmer, M Pusch, F Conti, K Imoto, S Numa
FEBS letters 293 (1-2), 93-96, 1991
5881991
Chloride/proton antiporter activity of mammalian CLC proteins ClC-4 and ClC-5
A Picollo, M Pusch
Nature 436 (7049), 420-423, 2005
5392005
Regions involved in the opening of CIC-2 chloride channel by voltage and cell volume
S Gründer, A Thiemann, M Pusch, TJ Jentsch
Nature 360 (6406), 759-762, 1992
4951992
Gating of the voltage-dependent chloride channel CIC-0 by the permeant anion
M Pusch, U Ludewig, A Rehfeldt, TJ Jentsch
Nature 373 (6514), 527-531, 1995
3961995
CLC chloride channels and transporters: structure, function, physiology, and disease
TJ Jentsch, M Pusch
Physiological reviews 98 (3), 1493-1590, 2018
3642018
Two physically distinct pores in the dimeric CIC-0 chloride channel
U Ludewig, M Pusch, TJ Jentsch
Nature 383 (6598), 340-343, 1996
3261996
Molecular determinants of KCNQ (Kv7) K+ channel sensitivity to the anticonvulsant retigabine
A Schenzer, T Friedrich, M Pusch, P Saftig, TJ Jentsch, J Grötzinger, ...
Journal of Neuroscience 25 (20), 5051-5060, 2005
3092005
Heteromultimeric CLC chloride channels with novel properties
C Lorenz, M Pusch, TJ Jentsch
Proceedings of the National Academy of Sciences 93 (23), 13362-13366, 1996
2841996
Multimeric structure of ClC‐1 chloride channel revealed by mutations in dominant myotonia congenita (Thomsen).
K Steinmeyer, C Lorenz, M Pusch, MC Koch, TJ Jentsch
The EMBO journal 13 (4), 737-743, 1994
2731994
Myotonia caused by mutations in the muscle chloride channel gene CLCN1
M Pusch
Human mutation 19 (4), 423-434, 2002
2652002
Properties of voltage‐gated chloride channels of the ClC gene family.
TJ Jentsch, W Günther, M Pusch, B Schwappach
The Journal of physiology 482 (suppl), 19-25, 1995
2631995
Surface expression and single channel properties of KCNQ2/KCNQ3, M-type K+ channels involved in epilepsy
M Schwake, M Pusch, T Kharkovets, TJ Jentsch
Journal of Biological Chemistry 275 (18), 13343-13348, 2000
2222000
The muscle chloride channel ClC-1 has a double-barreled appearance that is differentially affected in dominant and recessive myotonia
C Saviane, F Conti, M Pusch
The Journal of general physiology 113 (3), 457-468, 1999
2201999
Mutations in dominant human myotonia congenita drastically alter the voltage dependence of the CIC-1 chloride channel
M Pusch, K Steinmeyer, MC Koch, TJ Jentsch
Neuron 15 (6), 1455-1463, 1995
2181995
Biophysical properties of acid-sensing ion channels (ASICs)
S Gründer, M Pusch
Neuropharmacology 94, 9-18, 2015
1952015
Conservation of chloride channel structure revealed by an inhibitor binding site in ClC-1
R Estévez, BC Schroeder, A Accardi, TJ Jentsch, M Pusch
Neuron 38 (1), 47-59, 2003
1932003
Functional and structural conservation of CBS domains from CLC chloride channels
R Estévez, M Pusch, C Ferrer‐Costa, M Orozco, TJ Jentsch
The Journal of physiology 557 (2), 363-378, 2004
1872004
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Статии 1–20